Today Is Copernicus’s 540th Birthday. You May Remember Copernicus As The Man Who Said “Hey, What

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Here are 17 jaw-dropping photos of space that show us just how small we really are:

This photo of the moon and Earth taken from the International Space Station.

A dwarf galaxy, about 11 million light-years away from us.

Earth as seen from the moon in 1968.

A cluster of stars, 20,000 light-years away from Earth.

The first flower grown in the International Space Station, photographed by astronaut Scott Kelly.

Saturn, seen through an infared filter.

These visible “loops” on the surface of the sun can reach up to 15 times the diameter of Earth in height.

The Northen Lights just North of Chicago, viewed from the International Space Station.

The Quintuplet Cluster, located 100 light-years from the center of our galaxy.

Pluto and one of its moons, Charon.

The Great Pyramids of Giza, seen from space.

Astronaut Bruce McCandless maneuvering, untethered, above Earth in 1984.

Galaxy NGC 6240, 400 million light-years away from Earth.

Palomar 12, a cluster of stars on the outskirts of the Milky Way.

The remnants of an exploded star.

New York City, seen from the International Space Station.

And the remains of a supernova whose explosion may have been seen almost 2,000 years ago by Chinese astronomers.

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Maslow

Ok, so I don’t know how I ended up here and woah!

they made

characters

for

every

single

element

of the

periodic

table!

And also they made this

and this

*new ship* 

There’s even a granny!

It’s like

superheros

(there’s a guy who looks like Hulk btw)

and humans

and there are

twins!!

And Bethoveen

THEY MADE THOR

And there’s also this which made me laugh

I can’t! 

(source)

Why do we not discuss clouds more?

I mean look at that. That’s water.

Flying water.

FLYING

FUCKING

WATER

LIKE WHAT THE FUCK, WHY DO WE EVER STOP TALKING ABOUT THIS

WHAT IS THIS

HOW IS THIS EVEN

AND NOW THE FLYING WATER IS EATING A MOUNTAIN

GOD DAMN, WHAT

Tilt-shift meets interstellar imagery, by St. Tesla, via Neatorama.

How the Geneva Drive (the mechanical step that makes the second hand on a clock work by turning constant rotation into intermittent motion) works.

Neuroscientist Discovers Potential New Source for Pain Inhibition

A UT Dallas scientist has found a new neurological mechanism that appears to contribute to a reduction in pain.

According to Dr. Ted Price, associate professor in the School of Behavioral and Brain Sciences, the discovery of neuroligin-2 as a cause exacerbating chronic pain is significant for the research community. Although the findings likely won’t immediately lead to new pain therapies, the findings offer a potential new therapeutic direction to investigate, he said.

Price’s research on the topic has recently been published online in Pain, the journal of the International Association for the Study of Pain.

The study focused on the body’s inhibitory networks — a series of biochemical reactions that decrease certain neurological activity, such as pain. Price said a great deal of previous research in this area has focused on the activity of the neurotransmitter GABA, a chemical released by nerve cells in the brain.

Normally, a GABA neurotransmitter acts to inhibit neuronal activity, such as pain. However, when pain becomes chronic there is strong evidence that a process called GABAergic plasticity can cause GABA to lose its inhibitory activity, sometimes making the pain even worse.

The source of these excitatory actions in neuronal circuits has been broadly attributed to chloride ions, but Price’s research has found another potential cause of GABAergic plasticity: synaptic adhesion molecules called neuroligin-2.

“From a basic science perspective, we’re really excited about it because it demonstrates that the types of GABAergic plasticity that can occur in the setting of chronic pain are more diverse than we’ve appreciated before,” he said.

Price, who heads the undergraduate research program in neuroscience in the school, focuses much of his research on understanding the neuroscience behind pain, particularly chronic pain. He said individuals with chronic pain typically don’t receive the pain-reduction benefits delivered by inhibitory systems. Instead, they often experience increased pain.

“When you hit your hand with a hammer, almost everybody has the same reflex reaction — that is, to rub your finger which, in turn, helps to reduce pain. The reason that works is because it increases GABAergic inhibition in the spinal cord,” Price said. “However, people who have chronic pain — if they do the same thing — find that rubbing it actually makes the pain worse. That’s because the GABAergic system loses its efficacy and, in fact, can become excitatory.”

Price said the research is another step in determining why the GABAergic system stops working correctly in some people and provides a second theory for what drives the system.

“Having two ideas and different models will allow us to determine what the therapeutic opportunities are — creating something that will change that back to normal. The lack of performance in the inhibitory system is very detrimental to those who are in chronic pain,” he said.

Price said the development of chronic pain is, in essence, one’s body “learning” something that is bad.

“It’s changing the way the body functions — it’s learning. That learning, in the case of chronic pain, is aberrant — it’s causing the situation to get worse. If we can figure out what that form of learning was, then we can potentially reverse it. Understanding that the GABAergic system changes during this form of learning potentially offers a new therapeutic avenue,” he said.